Abstract
Neurotransmitter system dysfunction may contribute to the borderline personality disorder traits of impulsive aggression and affective instability. This article reviews evidence from neurochemical assays, receptor-density studies, neuroendocrine-challenge paradigms, functional neuroimaging studies, and candidate-gene research, which converge to identify particular neurotransmitter systems that seem to be dysregulated in patients with borderline personality disorder.
MeSH terms
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Acetylcholine / metabolism
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Affect*
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Aggression*
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Animals
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Arginine Vasopressin / metabolism
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Borderline Personality Disorder / metabolism*
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Borderline Personality Disorder / physiopathology
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Brain / metabolism*
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Humans
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Impulsive Behavior / metabolism
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Neurotransmitter Agents / metabolism*
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Norepinephrine / metabolism
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Serotonin / metabolism
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gamma-Aminobutyric Acid / metabolism
Substances
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Neurotransmitter Agents
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Arginine Vasopressin
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Serotonin
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gamma-Aminobutyric Acid
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Acetylcholine
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Norepinephrine