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Practice

A case of valproate-induced hyperammonemic encephalopathy: look beyond the liver

Saleh Alqahtani, Paolo Federico and Robert P. Myers
CMAJ September 11, 2007 177 (6) 568-569; DOI: https://doi.org/10.1503/cmaj.061272
Saleh Alqahtani MD
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Paolo Federico MD
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Robert P. Myers MD
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    Figure 1: Electroencephalogram taken on day 1 after the patient was admitted to hospital for the treatment of valproate-related hyperammonemic encephalopathy. Note the diffuse high-amplitude generalized slowing, consistent with diffuse cerebral dysfunction. Note: C = central, F = frontal, FP = frontpolar, O = occipital, T = temporal. Odd and even numbers in the electrode locations refer to the left and right hemispheres respectively.

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    Figure 2: Electroencephalogram taken on days 4 and 7 after the patient was admitted to hospital: (A) High-amplitude slowing has largely disappeared, and normal 10–11-Hz α waves are becoming apparent in the posterior head regions, suggestive of neurologic improvement. (B) Electroencephalogram appears almost completely normal, with well-developed α waves in the posterior head regions (arrows). Note: C = central, F = frontal, FP = frontpolar, O = occipital, T = temporal. Odd and even numbers in the electrode locations refer to the left and right hemispheres respectively.

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Canadian Medical Association Journal: 177 (6)
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Vol. 177, Issue 6
11 Sep 2007
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A case of valproate-induced hyperammonemic encephalopathy: look beyond the liver
Saleh Alqahtani, Paolo Federico, Robert P. Myers
CMAJ Sep 2007, 177 (6) 568-569; DOI: 10.1503/cmaj.061272

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A case of valproate-induced hyperammonemic encephalopathy: look beyond the liver
Saleh Alqahtani, Paolo Federico, Robert P. Myers
CMAJ Sep 2007, 177 (6) 568-569; DOI: 10.1503/cmaj.061272
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