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Research

Mortality attributable to nosocomial Clostridium difficile–associated disease during an epidemic caused by a hypervirulent strain in Quebec

Jacques Pépin, Louis Valiquette and Benoit Cossette
CMAJ October 25, 2005 173 (9) 1037-1042; DOI: https://doi.org/10.1503/cmaj.050978
Jacques Pépin
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Louis Valiquette
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Benoit Cossette
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Data supplements

  • Mortality attributable to nosocomial Clostridium difficile–associated disease during an epidemic caused by a hypervirulent strain in Quebec - Abridged Version

    Files in this Data Supplement:

    • Abridged Version PDF - Abridged Version (as appeared in the print version of CMAJ on October 25, 2005)

Related Articles

  • (2005). Highlights of this issue • Dans ce numéro. CMAJ, 173(9), 1013. Accessed December 07, 2019. Retrieved from http://www.cmaj.ca/content/173/9/1013.
  • Eggertson, L. (2005). C. difficile may have killed 2000 in Quebec: study. CMAJ, 173(9), 1020-1021. Accessed December 07, 2019. https://doi.org/10.1503/cmaj.051226.
  • Louie, T. J. (2005). How should we respond to the highly toxogenic NAP1/ribotype 027 strain of Clostridium difficile?. CMAJ, 173(9), 1049-1050. Accessed December 07, 2019. https://doi.org/10.1503/cmaj.051212.
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Canadian Medical Association Journal: 173 (9)
CMAJ
Vol. 173, Issue 9
25 Oct 2005
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Mortality attributable to nosocomial Clostridium difficile–associated disease during an epidemic caused by a hypervirulent strain in Quebec
Jacques Pépin, Louis Valiquette, Benoit Cossette
CMAJ Oct 2005, 173 (9) 1037-1042; DOI: 10.1503/cmaj.050978

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Mortality attributable to nosocomial Clostridium difficile–associated disease during an epidemic caused by a hypervirulent strain in Quebec
Jacques Pépin, Louis Valiquette, Benoit Cossette
CMAJ Oct 2005, 173 (9) 1037-1042; DOI: 10.1503/cmaj.050978
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  • Highlights of this issue • Dans ce numéro
  • C. difficile may have killed 2000 in Quebec: study
  • How should we respond to the highly toxogenic NAP1/ribotype 027 strain of Clostridium difficile?
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  • PubMed
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Cited By...

  • A small-molecule antivirulence agent for treating Clostridium difficile infection
  • Risk Factors for Acquisition and Loss of Clostridium difficile Colonization in Hospitalized Patients
  • Clostridium difficile PCR Ribotype 018, a Successful Epidemic Genotype
  • Decreasing Clostridium difficile Infections by an Antimicrobial Stewardship Program That Reduces Moxifloxacin Use
  • Epidemic Clostridium difficile Strains Demonstrate Increased Competitive Fitness Compared to Nonepidemic Isolates
  • Role of Leptin-Mediated Colonic Inflammation in Defense against Clostridium difficile Colitis
  • Variations in Virulence and Molecular Biology among Emerging Strains of Clostridium difficile
  • Nontoxigenic Clostridium difficile Protects Hamsters against Challenge with Historic and Epidemic Strains of Toxigenic BI/NAP1/027 C. difficile
  • Characterizations of Clinical Isolates of Clostridium difficile by Toxin Genotypes and by Susceptibility to 12 Antimicrobial Agents, Including Fidaxomicin (OPT-80) and Rifaximin: a Multicenter Study in Taiwan
  • Clinical and Laboratory Characteristics of Clostridium difficile Infection in Patients with Discordant Diagnostic Test Results
  • The effect of hospital-acquired infection with Clostridium difficile on length of stay in hospital
  • Lack of Association between Clinical Outcome of Clostridium difficile Infections, Strain Type, and Virulence-Associated Phenotypes
  • Prophage-Stimulated Toxin Production in Clostridium difficile NAP1/027 Lysogens
  • Neutralization of Clostridium difficile Toxin A with Single-domain Antibodies Targeting the Cell Receptor Binding Domain
  • The Changing Epidemiology of Clostridium difficile Infections
  • Efficacy of cleaning products for C difficile: Environmental strategies to reduce the spread of Clostridium difficile-associated diarrhea in geriatric rehabilitation
  • Multilocus Sequence Typing of Clostridium difficile
  • Antiapoptotic Proteins Bcl-2 and Bcl-XL Inhibit Clostridium difficile Toxin A-Induced Cell Death in Human Epithelial Cells
  • Effects of Subinhibitory Concentrations of Antibiotics on Colonization Factor Expression by Moxifloxacin-Susceptible and Moxifloxacin-Resistant Clostridium difficile Strains
  • Motility and Flagellar Glycosylation in Clostridium difficile
  • Antimicrobial resistance in hospitals: How concerned should we be?
  • Effects of Exposure of Clostridium difficile PCR Ribotypes 027 and 001 to Fluoroquinolones in a Human Gut Model
  • Fairness of financial penalties to improve control of Clostridium difficile
  • Clostridium difficile Infections in a Canadian Tertiary Care Hospital before and during a Regional Epidemic Associated with the BI/NAP1/027 Strain
  • Rifampin and Rifaximin Resistance in Clinical Isolates of Clostridium difficile
  • Tolevamer, an Anionic Polymer, Neutralizes Toxins Produced by the BI/027 Strains of Clostridium difficile
  • Increased Sporulation Rate of Epidemic Clostridium difficile Type 027/NAP1
  • Use of Highly Discriminatory Fingerprinting to Analyze Clusters of Clostridium difficile Infection Cases Due to Epidemic Ribotype 027 Strains
  • The Changing Spectrum of Clostridium Difficile Associated Disease: Implications for Dentistry
  • Yield of Stool Culture with Isolate Toxin Testing versus a Two-Step Algorithm Including Stool Toxin Testing for Detection of Toxigenic Clostridium difficile
  • Auto-catalytic Cleavage of Clostridium difficile Toxins A and B Depends on Cysteine Protease Activity
  • Diarrhoea associated with antibiotic use
  • tcdC Genotypes Associated with Severe TcdC Truncation in an Epidemic Clone and Other Strains of Clostridium difficile
  • Molecular Analysis of Clostridium difficile PCR Ribotype 027 Isolates from Eastern and Western Canada
  • Measuring the presence of chronic diseases.
  • How should we respond to the highly toxogenic NAP1/ribotype 027 strain of Clostridium difficile?
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