EditorialOn the etiology of vitiligo and gray hair
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Cited by (259)
Melatonin: A Cutaneous Perspective on its Production, Metabolism, and Functions
2018, Journal of Investigative DermatologyCitation Excerpt :Therefore, testing of topically applied melatonin during defined circadian windows as an external modulator of intracutaneous clock activity is warranted (Slominski et al., 2015). Finally, in light of the autodestruction and oxidative stress theories of vitiligo pathogenesis (Lerner, 1971; Schallreuter et al., 2008), melatonin and serotonin might play a protective role in vitiligo pathogenesis (see viewpoint 4 in Schallreuter et al., 2008). This is further supported by the low expression of TPH1 in vitiligo (Schallreuter et al., 2012).
Nonsegmental vitiligo update
2016, Dermatologica SinicaCitation Excerpt :Vitiligo is an acquired depigmenting disorder that should be differentiated from congenital hypopigmentation such as albinism, piebaldism, dyschromatosis symmetrica hereditaria, and nevus depigmentosus.1–3 Idiopathic guttate hypomelanosis, tinea versicolor, and chemically induced leukoderma caused by phenols, catechols, and rhododendrol should also be distinguished from authentic vitiligo.1,3–5 Segmental vitiligo affecting a unilateral dermatome-like cutaneous segment is less frequent than nonsegmental or generalized vitiligo.6
Melanocytes and VitiLigo (and Hair Graying)
2014, Pathobiology of Human Disease: A Dynamic Encyclopedia of Disease MechanismsUnderstanding of Pathomechanisms and Clinical Practice for Vitiligo
2023, Annals of DermatologyEffect of vitiligo on auditory functions: Is there any association?
2021, International Archives of OtorhinolaryngologyInvolvement of adenylate cyclase/cAMP/CREB and SOX9/MITF in melanogenesis to prevent vitiligo
2021, Molecular and Cellular Biochemistry
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From the Section of Dermatology, Yale University School of Medicine, New Haven, Connecticut 06510.